Optic neuritis

Optic neuritisEtiology: common acute and chronic infections (influenza, tonsillitis, typhoid fever, syphilis, tuberculosis, brucellosis and other) and alcohol (methyl alcohol, and others), diabetes, gout, blood disorders, nephritis, focal infection (tonsillitis, sinusitis, otitis media, and others), inflammatory diseases of the brain and its membranes, etc. Pathogenesis: the immediate spread of the inflammatory process in the optic nerve of the sinuses or shells brain metastasis of infecta with bacteremia, the response of neural tissue to sensitisation by common infections and intoxications. Symptoms. The lower view, concentric, sometimes irregular narrowing of the field of view, primarily in color, Central and paracentral scotomas in the visual field. The optic nerve disc hyperemia, not prominere (with the exception of the form of nephritis with edema), its borders are fuzzy. The retinal arteries is usually slightly narrowed, veins dilated and tortuous. Possible bleeding on the disk and about him. From congestion of the optic nerve and pseudonigrita differs coming fast and significant decrease in visual function. Treatment first of all the underlying disease; the use of antibiotics (better broad-spectrum) and corticosteroids; while the phenomena of swelling of 50% solution of glycerin inside (at the rate of 1.5 g/kg) during the week, diacarb 0.25 g 2-3 times a day, 40% glucose solution 20 ml/15-20 injections, 40% solution of hexamethylenetetramine (urotropine)/10 ml-10 injections, vitamins B1 and B12/m 1 ml daily 15 to 20 injections. In the subacute period, inflammatory phenomena - General restorative, tonic, and vasodilator drugs (nicotinic acid, 0.05 g, yexin 0.25 g 2 times a day, papaverine 0.02 g 3 times a day, aloe extract, phibbs, and others), autohemotherapy. The forecast. Timely treatment helps preserve high enough view. Late treatment usually occurs optic nerve atrophy. The prognosis is worse in toxic lesions of the optic nerve..

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